Welcome to the Journal of Neurology & Neuromedicine

Manuscript Guidelines

The journal has specific rules to formatting a manuscript that authors should adhere to before shipping their manuscript. These guidelines are primarily intended to make the submission of manuscript quick and easy.    Read More

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Ethics & Disclosures

Journal of Neurology and Neuromedicine is primarily based on values centered on loyalty, commitment, scientific accuracy, and ethics. It has adopted clear and rigorous ethical guidelines for best working practices.    Read More

What happens next to your Submission

Each article we publish benefits from hundreds of hours of work by Chief editors, Sectional editors, Reviewers, Manuscript editors, Proofreaders, Graphics and Web experts, who work to ensure that the manuscript meets our standards. Read More

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Focus & Scope


The overarching goal of the Journal of Neurology and Neuromedicine is to remain as a credible source of neurological sciences based information encompassing a variety of relevant areas such as clinical studies, cellular and molecular studies, disease mechanisms, diagnostic approaches, epidemiology, medical aspects, computational studies and treatment options of the most common neurobiological complexities with an emphasis on research that is genetically, structurally, physiologically and pathophysiologically relevant.

The integral part of our scholarly mission is to ensure the accuracy of journal quality in accord with the highest standards of professional ethics.

“Quality is our Priority.
It is the Foundation of our Publication

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Recent Articles


Vol 3-6 Mini Review

Neurotransmitters and their Receptors as the Upstream Regulators of the Most Common Human Cancers and their Stem Cells

Hildegard M. Schuller

Department of Biomedical and Diagnostic Sciences, College of Veterinary Medicine, University of Tennessee, Knoxville TN, USA

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Vol 3-6 Commentary

Novel Modality of GSK-3 Inhibition For Treating Neurodegeneration

Ido Rippin, Hagit Eldar-Finkelman*

Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Israel

DOI: 10.29245/2572.942X/2018/6.1227 View / Download Pdf View Full Text
Vol 3-6 Mini Review

Vagal-Immune Interactions in the Control of Hypertension

Renata M. Lataro1, Helio C. Salgado2*

1Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina, Florianópolis, Santa Catarina, Brazil

2Department of Physiology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, São Paulo, Brazil

There is a consensus in the literature that the autonomic dysfunction in arterial hypertension involves the increase of sympathetic activity and, also, reduction of vagal tone. In the last years, the role played by inflammation in the development of hypertension and target organs injuries, such as heart and blood vessels, has been emphasized. Although the clinical importance of sympathetic hyperactivity and its treatment of arterial hypertension is recognized, the therapeutic benefit of increasing parasympathetic activity in hypertensive patients still requires an in-depth investigation. The increased vagal activity may produce beneficial effects on cardiovascular modulation and inflammation, preventing target organ damage. Parasympathetic neurotransmission can be improved by the inhibition of acetylcholinesterase action. Anticholinesterase agents prevent the hydrolysis of acetylcholine by acetylcholinesterase, prolonging its availability within the cholinergic cleft. This article will highlight the key concepts of the cardiac autonomic imbalance and the increase of acetylcholine availability under inflammation and control of arterial hypertension. In conclusion, significant evidence exists associating the reduction of parasympathetic activity and the occurrence of inflammation involving the pathophysiology of hypertension; suggesting that the improvement of vagal activity by the increased availability of acetylcholine has a remarkable potential for the therapeutic intervention on arterial hypertension.

DOI: 10.29245/2572.942X/2018/6.1226 View / Download Pdf View Full Text
Vol 3-6 Opinion Article

Persistent Antigens Hypothesis: The Human Leukocyte Antigen (HLA) Connection

Lisa M. James1,2,3, Apostolos P. Georgopoulos1,2,3,4*

1Brain Sciences Center, Department of Veterans Affairs Health Care System, Minneapolis, MN, 55417, USA

2Department of Neuroscience, University of Minnesota Medical School, Minneapolis, MN 55455, USA

3Department of Psychiatry, University of Minnesota Medical School, Minneapolis, MN 55455, USA

4Department of Neurology, University of Minnesota Medical School, Minneapolis, MN 55455, USA

DOI: 10.29245/2572.942X/2018/6.1235 View / Download Pdf View Full Text
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